Oral Health and Dental Management

Relation to peri-implantitis, bacteria and host immune factor

10^th Dentists and Prosthodontics Annual Meeting

June 27-28, 2016 New Orleans Louisiana, USA

G Rutger Persson

University of Washington, USA

Posters & Accepted Abstracts: Oral Health Dent Manag

Abstract:

The objective of the presentation is to provide a comprehensive summary of current perceptions with focus on the infectious etiology and factors that may enhance the risk for infection resulting in implant mucositis, or peri-implantitis. The etiology of peri-implantitis is multifactorial. The oral microbiota is not only diverse, it comprises of a dense population of microorganisms including both aerobic and anaerobic bacterial species ranging from obligate to facultative aerobic or anaerobic species. As soon as a dental implant is exposed in the oral cavity, microorganisms will start to colonize the exposed dental implant surface. Dental implant design and surface chemistry may have an impact on the invasion of oral microorganisms into the fixture-abutment interface. Bacteria usually not associated with periodontitis have, however, also been found in PICF at implants with peri-implantitis forming a cluster of pathogens with Treponema socranskii, Staphylococcus aureus, Staphylococcus anaerobius, Streptococcus intermedius, and Streptococcus mitis comprising 30% of the total microbiota. The occurrence of failing dental implants has been associated with low serum antibody titers and with low avidity levels to S. aureus. Profuse bleeding and/or suppuration in untreated peri-implantitis can be associated with higher concentrations of interleukin-1β, IL-8, tumor necrosis factor (TNF)-α and vascular endothelial growth factor VEGF in peri-implant crevicular fluid. the profile of inflammatory cytokines in implant crevicular fluid represents innate immune responses. Studies on successful treatment of implant mucositis, or periimplantitis using traditional non-surgical debridement, laser therapy, antiseptics, and antibiotics have shown that have so far not allowed the development of predictable treatment modalities of inflammatory conditions of dental implant surrounding tissues.

Biography :

Email: rutger.persson@hkr.se